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The magnitude of Chac1 downregulation exceeded that of canonical refeeding-responsive genes controlling hepatic glucose metabolism, such as the downregulation of G6pc or upregulation of Gck (Figure 6A), indicating robust transcriptional suppression of Chac1 during feeding

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GHK-Cu verstrkt auch die Aktivierung von Metalloproteinasen (MMPs), die am Abbau beschdigten Gewebes und der Frderung der Neubildung von Gewebe beteiligt sind

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