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In the context of severe bleeding, increasing levels of O-GlcNAcylation lead to a reduction in acute inflammation by attenuating the increase in pro-inflammatory cytokines, such as TNF or IL-6, as has been shown after trauma in animals in which O-GlcNAcylation was stimulated [52, 54, 70]

Within this concept FOXO factors antagonize classical growth factor signaling promoting repair processes

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