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[54] The inhaled substances trigger chemical reactions in nerve endings in the brain due to being similar to naturally occurring substances such as endorphins and dopamine, which are associated with sensations of pleasure

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42 In addition, CCL2-CCR2 signaling activates CCL3-CCR1 signaling in MAMs, which support MAM accumulation at the metastatic site, 59 which enhances signaling pathway-mediated metastatic cell growth via the FMS-like tyrosine kinase 1-focal adhesion kinase (FAK1)-CSF1 and CSF1-C-ets-2-microRNAs pathways in macrophages

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