Conclusions A series of mechanisms can potentially explain how GLP-1RAs may contribute to reduce the risk of stroke and promote brain protection in patients with T2DM (Fig
CD36 favors uptake of palmitic acid and sensitivity of aggressive breast cancer cells to ferroptosis To investigate the role of CD36 in the PA-induced ferroptosis in TNBC, SUM-159 and SUM-159-CD36 cells were exposed to PA, erastin-2, or RSL3 to evaluate the accumulation of peroxidized lipids
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