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This dual action may involve special neurons in the hypothalamus that are only activated when both hormones are present simultaneously

This leads to reduced production of malonyl-CoA and consequently relieves the inhibition of carnitine palmitoyltransferase 1 (CPT1), promoting fatty acid -oxidation

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5) By modulating this pathway, it may be possible to alleviate the detrimental effects of elevated UA levels on endothelial function and reduce associated cardiovascular risks

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