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A recent analysis using Mendelian randomization showed that genetically predicted homocysteine levels increased the odds of NAFLD.[ref] Related article: NAFLD genes Cardiovascular disease: To answer the question of whether a modestly elevated homocysteine level is causally associated with an increased risk of cardiovascular disease, researchers again used huge genetic data sets and Mendelian randomization to determine causality

BPC-157 addresses this connection at the cellular level

This arrangement places GHK-Cus copper-binding strength in the same general range as the labile copper pool carried by albumin in plasma which is precisely why the peptide can plausibly participate in physiological copper exchange rather than either locking copper away permanently or dumping it uncontrollably