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Similarly, intestinal organoid systems rely on ITS or B-27 as basal support alongside niche specific factors (Wnt3a, R spondin 1, Noggin) to sustain Lgr5+ stem cell compartments [7]

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They are gut-hormonebased metabolic drugs first approved by the FDA in 2005 (exenatide) for type 2 diabetes, now widely used for obesity

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The result is a discrete pulse of GH release subject to normal somatostatin-mediated negative feedback, theoretically preserving pulsatility, unlike exogenous recombinant GH which produces sustained non-physiologic serum levels