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In conclusion, GLP-1 receptor agonists effectively alleviate hepatic inflammation in MASLD by suppressing pro-inflammatory cytokine release, inhibiting NF-B signaling, modulating the phenotypes of immune cells, and reducing the activation of inflammasomes

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Carnitine-mediated acyl transfer not only ensures efficient fatty acid utilization and energy production in mitochondria and peroxisomes but also prevents the toxic accumulation of VLCFAs in cells

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