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For antineoplastics, the reduction in plasma concentration is linked to the induction of the CYP1A2 isoform by smoking by the same mechanism

Furthermore, cigarette smoke can activate the autophagy-dependent mechanism mediated by the deacetylase HDAC6, while causes the cilia proteins to be delivered to the lysosome for degradation or recycling, shortening the airway cilia ( in vitro to cause e-cadherin-mediated barrier function destruction of airway epithelial cells, leading to increased infection risk by viruses and bacteria that exacerbate COPD (Oldenburger et al., 2014)

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ResultsOverexpression of miR-320d reduces CXCL8 and IL-1 levels in non-COPD-derived PBECs which is compromised in COPD-derived PBECs We first investigated whether the suppressive effects of miR-320d overexpression on pro-inflammatory cytokine expression differ between non-COPD and COPD-derived PBECs