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KET is the prototypical non-competitive antagonist at N -methyl-D-aspartate glutamate receptors and a clinically approved anesthetic
Under aerobic condition, SOD overexpression boosted the growth of S-3/pKLH167, S-3/pKLH337, S-3/pKLH341, and S-3/pKLH344 with 1 mmol/L H 2 O 2 treatment compared with the control strain S-3/pKLH32 (Figure 4A)
This could further indicate a shift towards attempting to synthesize EGT in the mutant, given that histidine is a key biosynthetic precursor of EGT
doi: 10.1007/s12035-024-03939-6 Cosentino F, Rubattu S, Savoia C, Venturelli V, Pagannonne E, Volpe M
Glucagon-like peptide-1 receptor agonist treatment attributes important to injection-experienced patients with type 2 diabetes mellitus: a preference study in Germany and the United Kingdom
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