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This suggests that the changes in glutathione metabolism in diabetes are probably mediated by non-glycemic mechanisms and these mechanisms may be more relevant in persons with microvascular complications

It implies that GSTP1 codon 105 polymorphism may be a marker for risk of wound complications microvascular reconstruction 37 , emphasizing its important role in wound healing

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This depletion of GSH results in a worsening effect in many disease models