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Finally, the potential enhancement mechanism of non-nicotinic constituents was investigated by nicotine metabolism and monoamine oxidase A (MAOA) activity inhibition in the striatum of mice and human recombinant MAOA

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In the acute phase, the TNBS-treated Tg(h GPR15 ) mice presented more severe intestinal inflammation than the Gpr15 +/+ mice did (Fig

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