Ishijima M, de Avila ED, Nakhaei K, Shi W, Lux R, Ogawa T
In some contexts it is suggested that it could support mitochondrial function or neuronal signalling, but the clinical evidence is heterogeneous and does not allow generalisation of benefits to the whole population
This depletion may reduce the production of vitamin K-dependent clotting factors, increasing the risk of bleeding and Supplementation with vitamin K might be necessary in patients on long-term anticonvulsant therapy to prevent deficiency-related issues
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