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Activation of MC1 receptors can darken skin (hence why its precursor was a tanning agent), and indeed using bremelanotide frequently can cause increased melanin production and hyperpigmentation (darkening of the skin) in some users

Nephroprotective effects of b-carotene on ACE gene expression, oxidative stress and antioxidant status in thioacetamide induced renal toxicity in rats

Its greatly offset by the cancer risk

supports the use of GLP-1s for the treatment of heart failure with preserved ejection fraction, perhaps through a similar mechanism [16]