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BSO is an inhibitor of GSH synthase that has been used to deplete GSH in vitro , including in macrophages ( We analyzed the gene expression profile and identified patterns of LPS-induced genes that were inhibited by endogenous GSH or that, on the contrary, required GSH for their induction

Current evidence from large-scale clinical trials and regulatory surveillance does not establish a causal link between GLP-1 receptor agonists and increased risk of venous thromboembolism (deep vein thrombosis or pulmonary embolism)

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