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While limited research exists on cytotoxic T cells (Tc) in MASLD, studies in other immune disorders suggest SIRT1 regulates Tc function through T cell receptor expression and deacetylation of effector proteins such as perforin and granzyme

As a complementary approach, rational prodrug designs and receptor-targeted conjugates should be explored to restrict activation to cells that overexpress disease-associated markers in the endometriotic microenvironment