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Importantly, not all variants confer increased risk: loss of function variants in HSD17B13, a lipid droplet-associated protein in hepatocytes, have been consistently associated with reduced hepatic inflammation and a lower risk of fibrosis and advanced liver disease [16]

This well-established approach recognises that the physiological mechanisms behind obesity and T2D are inextricably intertwined, and thus recommends treating obesity not merely as a risk factor but as the primary therapeutic target in T2D

Joel Habener, Dr

In injured organs, the repair process is often unsatisfactory because of tissue defects, chronic inflammation accumulation and other abnormal pathologies