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One plausible explanation is that fatty acids of various carbon chain lengths may regulate the conformational changes and transcriptional activities of PPARs in different or even opposing manners [26]

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The consequences of this oxidative damage extend far beyond simple tiredness

Further exploring their role under abiotic stresses, gpx3 (H 2 O 2 -sensitive mutant) of Saccharomyces cerevisiae were complemented with the GhGPXs, and it suggests that GhGPXs have similar function to GPX3 in yeast, revealing their participation in the oxidative stress response

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