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GLP-1 acts through binding to its receptor (GLP-1R), triggering a downstream signaling cascade able to induce a potent stimulation of glucose stimulated insulin secretion (GSIS) in -cells, as well as inhibition of -cell glucagon release

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Thus, immunoneutralization with antibodies against GIP significantly inhibited oral-glucose-stimulated insulin secretion ( The interest now focussed on gut glucagon ( On the other hand, it was also clear that glicentin did not represent the full proglucagon molecule