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The fundamental mechanism is the activation of phase II detoxification enzymes or antioxidase controlled by Nrf2/ARE, including the expression of NAD(P)H quinone dehydrogenase 1 (NQO1), GPX, and superoxide dismutase (SOD) (Truong et al., 2014)

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Particularly, the activation of adaptive stress response pathways by AQ-QI and DEAQ-QI was elucidated using high-content imaging-based BAC-GFP toxicity assays

For experimental synchronization and amplification, worms were transferred to fresh plates and grown at 20 C