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GLP-1s may not cure Alzheimers as a monotherapy, but they are reshaping how we think about the interface of neurodegenerative disease etiology, metabolic health, vascular function and they likely will cement themselves as foundational tools in delaying cognitive decline across aging populations

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GIP enhances insulin secretion, may improve fat metabolism, and influences energy expenditure

In clinical practice, we have found this best to be introduced toward the end of treatment to avoid symptom reactions