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Different types of genetic variants may disrupt the circuits of social interactions and repetitive behaviours, resulting in a complex matrix of genes, synapses, circuits, and behaviours

In those, loss of NRF2 resulted in a more rapid onset and more severe clinical course of the EAE model, which was accompanied by increased glial activation, exacerbated spinal cord damage, and axonal degeneration as well as increased levels of proinflammatory cytokines (Rehman et al., 2019)

[2] [3] Chemically, Semax is a peptide and a synthetic analogue of a fragment of adrenocorticotropic hormone (ACTH)

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Dawczynski, M