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Figure 3 Elevated GPX4 and Ferrous Iron Are Potential Therapeutic Targets of CRC Persister Cells and Tumors The key event in ferroptosis is cell membrane damage associated with peroxidation of lipids ( GPX4 expression

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6eg), which is readily accessible in the prefusion conformation on the surface of the virus and could therefore act as a decoy for the immune system while vulnerable epitopes are shielded by glycosylation (Extended Data Fig

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