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Indeed, with an estimated prevalence of 1 in 20006000 live births, 22q11DS is the most common recurrent pathogenic deletion [1, 2]

The peptide contains a disulfide bridge between two cysteine residues, which stabilizes its three-dimensional structure

The mechanism is hypothesized to involve enhanced Schwann cell function and axonal sprouting, potentially via the growth factor pathways already implicated in its other activities

This peptide plays a key role in reducing inflammation, which is crucial for faster recovery from bone fractures and injuries

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